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Atenfar (Atorvastatin)

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Atenfar is a medication prescribed by a doctor to lower cholesterol in people who have been diagnosed with high cholesterol (high levels of cholesterol in the blood). Doctors diagnose high cholesterol through a simple blood test. Cholesterol (and triglycerides) are fats that are made in your body. While some cholesterol is necessary for the body, too much cholesterol is dangerous to your health. Cholesterol, specifically, is made in the liver. Lowering "bad" cholesterol and triglycerides and raising "good" cholesterol decreases the risk of heart disease and helps prevent strokes and heart attacks This drug can also lower the risk for heart attack or stroke in patients with diabetes.

Other names for this medication:
Alvastin, Ampliar, Anzitor, Ateroclar, Atocor, Atoris, Atorvastatin, Atorvin, Biger, Caduet, Colostat, Holisten, Kolestor, Liparex, Lipidan, Lipidra, Lipobi, Lipodial, Lipofin, Liponorm, Lipovastatin, Livas, Prevencor, Storvas, Taven, Torid, Torivas, Torva, Torvazin, Totalip, Trova, Vastina, Xelitor, Zarator

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Trileptal, Astelin, Lunesta, Imovane, Starlix, Fasigyn, Persantine, Ferodr, Dolodol, Cyplan, Conquet, Teliprolol, Carvibeta,

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Also known as: Atorvastatin


Atenfar (atorvastatin) belongs to a group of drugs called HMG CoA reductase inhibitors, or "statins."

Atenfar is used together with diet to lower blood levels of "bad" cholesterol (low-density lipoprotein, or LDL), to increase levels of "good" cholesterol (high-density lipoprotein, or HDL), and to lower triglycerides (a type of fat in the blood).

Atenfar is used to treat high cholesterol, and to lower the risk of stroke, heart attack, or other heart complications in people with type 2 diabetes, coronary heart disease, or other risk factors such as genetically high cholesterol.

Atenfar is for use in adults and children who are at least 10 years old.


Generic Atenfar can be taken in tablets. You should take it by mouth. It is better to take Generic Atenfar once a day at the same time with meals or without it. If you want to achieve most effective results do not stop taking Generic Atenfar suddenly.


If you overdose Generic Atenfar and you don't feel good you should visit your doctor or health care provider immediately.


Store at room temperature between 20 to 25 degrees C (68 to 77 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Atenfar are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


You should not use Atenfar if you are allergic to atorvastatin, or if you have:

liver disease; or if you are pregnant or breastfeeding.

Atorvastatin can harm an unborn baby or cause birth defects. Do not use if you are pregnant. Stop taking Atenfar and tell your doctor right away if you become pregnant. Use effective birth control to prevent pregnancy while you are taking this medicine.

Atorvastatin may pass into breast milk and could harm a nursing baby. Do not breastfeed while you are taking Atenfar.

To make sure this medicine is safe for you, tell your doctor if you have ever had:

liver problems; muscle pain or weakness; kidney disease; diabetes; a thyroid disorder; or if you drink more than 2 alcoholic beverages daily.

Atenfar can cause the breakdown of muscle tissue, which can lead to kidney failure. This happens more often in women, in older adults, or people who have kidney disease or poorly controlled hypothyroidism (underactive thyroid).

Atorvastatin is not approved for use by anyone younger than 10 years old.

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Effect of a topical herbal cream on the pain and stiffness of osteoarthritis: a randomized double-blind, placebo-controlled clinical trial. A panel of murine monoclonal antibodies (MAbs) against porcine growth hormone (pGH) has been raised from BALB/c mice. The protein biomarkers identified are prominently observed in the time-of-flight mass spectra (TOF MS) of bacterial cell lysate supernatants ionized by matrix-assisted laser desorption/ionization (MALDI). After DNA extraction, TSPY gene was sought as a surrogate of Y chromosome. Anal sphincter spasm and megacolon are pathophysiologic abnormalities associated with pediatric constipation-encopresis. All patients presented no evidence of disease in the abdominal cavity but retroperitoneal metastasis, which was the unique metastatic site. The objective of this study was to determine whether AAA progressed during protracted AngII infusion. These complications are described separately in the literature as being associated with chorioangioma of the placenta, but rarely appear together in a single case. Control of fecal incontinence: continued success with the Malone procedure. Piperazine-1,4-diium pyridine-2,3-dicarboxyl-ate methanol monosolvate.

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Pretreatment of donor lungs with ATL-1223 was as efficacious as other treatment strategies in protecting against ischemia-reperfusion injury. Urine leakage from the hydronephrotic sac could be avoided because the puncture site was sealed firmly between the two adjacent balloons at the tip of the catheter. Five patients with influenza-associated encephalitis/encephalopathy and sudden-onset neurologic symptoms following a prodromal flulike episode were examined by MR and diffusion-weighted imaging (DWI). Degradation of 2,4-dinitrophenol by free and immobilized cells of Rhodococcus erythropolis HL PM-1. Moreover, we identify a specific BARD1 isoform, which might act as tumor diagnostic and prognostic markers. Tests of pulmonary mechanical function may be used in determining the prominent site of pulmonary reaction to intervention. All patients were investigated for the presence of RBC autoantibodies and alloantibodies, followed by antibody identification for positive patients. ON THE CRIMINAL ACTIVITY OF FASHIST PHYSICIANS DURING THE WORLD WAR II. These data suggest that either p53 can induce apoptosis through a transcriptionally independent mechanism, a function lost by p53175P, or that zopiclone show up in drug test this mutant has specifically lost the ability to activate genes which contribute to cell death, despite activation of genes responsible for the G1 arrest.

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